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The introduction of PET imaging with AV-1451 (flortaucipir) has provided a unique
insight into the pathophysiology of Alzheimer Disease (AD). Prior work has consistently
found elevated levels of flortaucipir binding in the lateral temporal lobes, precuneus,
and lateral parietal regions in individuals with preclinical and clinical AD. It is
unknown whether this tracer is sensitive to primary age-related increases in tau in
the medial temporal lobe and whether off-target binding in the choroid plexus and
basal ganglia vary as a function of age.
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