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Determining early tau accumulation is important for studying aging and AD, and is
only as accurate as the signal to noise ratio of the tracer. Along with tau, [18F]-AV-1451 has been reported to bind to neuromelanin, MAO, iron, lepto-meningeal melanocytes,
and microhemorrhages. [18F]-AV-1451 successfully differentiates healthy controls (HCs) from ADs, however we
wanted to explore off-target binding and the variability seen in HCs expected to have
no tau accumulation (Figure 1).
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