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Patients with clinical probable dementia with Lewy bodies (DLB) commonly have β-amyloid
pathology in addition to Lewy body disease. Understanding the influence of β-amyloid
deposition on clinical disease progression in DLB is critical for prognosis and targeting
underlying neurodegenerative pathologies. However, it has been shown that the rate
of β-amyloid accumulation vs baseline SUVR follows an inverted U-shape among normal
to clinical AD dementia subjects; but, the nature of this relationship is unknown
in DLB. Our objective was to determine the rate of longitudinal β-amyloid accumulation
by PET vs baseline SUVR in DLB compared to controls, and the association between β-amyloid
accumulation and disease progression in DLB.
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