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Accumulation of cerebral beta-amyloid (Aβ) is a hallmark of Alzheimer’s Disease (AD).
The widespread distribution of Aβ in late AD stages is well-established, however the
regional progression of Aβ deposition in early AD remains uncertain. We studied regional
Aβ deposition in cognitively normal (CN) individuals from the Baltimore Longitudinal
Study of Aging using multivariate pattern analysis techniques and nonlinear mixed-effects
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