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Down syndrome (DS) is frequently associated with the presence of comorbid beta-amyloid
(Aβ) and neurofibrillary tau tangles, both of which are the signature hallmarks of
Alzheimer’s disease (AD). Clarifying the coupling between both neuropathologies may
provide further insights into the disease mechanisms and inform optimisation of clinical
trials in DS. However, the in vivo relationships between Aβ and tau remains to be delineated in people with DS.
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