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Characterising biomarker trajectories during the presymptomatic phase of Alzheimer’s
disease is critical to identifying optimal windows for early treatment. Accumulation
of β-amyloid plaques is an early change in the disease and can be assessed quantitatively
in vivo using amyloid-specific PET tracers. These measures are a potential outcome measure
for secondary prevention studies, but they can be affected by numerous factors, including
age, genetics, vascular changes, and analysis technique. We examined longitudinal
changes in amyloid accumulation in a large sample of healthy individuals from a British
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